Advances in Virus Research
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Table of contents
- Contentsv
- Chapter 1: Viruses, Vectors, and Vegetation: An Autobiography1
- I. From Childhood in Europe to America1
- II. Brooklyn Botanic Garden5
- III. Virus Nomenclature and Classification13
- IV. Cold Spring Harbor14
- V. The Cadang-Cadang Disease16
- VI. Dark Clouds on the Horizon17
- VII. Insect Viruses and Cell Culture17
- VIII. Electron Microscopy21
- IX. Books22
- X. International Connections24
- Acknowledgments26
- References27
- Books29
- Chapter 2: Honey Bee Viruses33
- I. Introduction34
- II. Common Honey Bee Viruses35
- A. Deformed wing virus35
- B. Sacbrood virus37
- C. Black queen cell virus38
- D. Kashmir bee virus39
- E. Acute bee paralysis virus41
- F. Chronic bee paralysis virus42
- III. Taxonomy43
- A. Virion properties43
- B. Genome organization and classification45
- IV. Transmission Modes49
- A. Horizontal transmission50
- B. Vertical transmission55
- C. Discussion56
- V. Pathogenesis58
- A. Causal relationship between a virus and a disease59
- B. Tissue tropism59
- C. Host range61
- VI. Host Defense Mechanisms63
- A. Colony-level defense64
- B. Individual-level defense66
- VII. Management of Virus Infections69
- VIII. Future Directions71
- Acknowledgments72
- References72
- Chapter 3: Use of Functional Genomics to Understand Influenza-Host Interactions81
- I. Introduction82
- II. Model Systems of Influenza A Infection Used in Functional Genomics83
- A. Cell culture models83
- B. Murine models86
- C. Nonhuman primate models90
- D. New diagnostic approaches93
- III. Conclusions95
- Acknowledgments
- References96
- Chapter 4: A Guide to Viral Inclusions, Membrane Rearrangements, Factories, and Viroplasm Produced D101
- I. Introduction103
- II. Viroplasm, Virosomes, Factories, and Inclusions104
- III. Membrane Rearrangements Occurring During the Replication of the Positive-Stranded RNA Viruses104
- A. Regulation of membrane traffic in the early secretory pathway106
- B. Picornavirus replication induces numerous membrane vesicles109
- C. Alphaviruses produce membrane invaginations and spherules117
- D. The Flaviviridae replicate in vesicular packets and membraneous webs119
- E. The Nidovirales replicate in association with double-membraned vesicles122
- IV. Virus Factories and Inclusion Bodies Generated by Large DNA Viruses124
- A. Cytoplasmic virus factories formed by large cytoplasmic DNA viruses124
- V. Herpesviruses Induce Nuclear Inclusions and Cytoplasmic Assembly Sites145
- A. Herpesviruses145
- B. Herpesvirus replication generates inclusions in the nucleus146
- C. Cytoplasmic inclusions form during late stages of herpesvirus tegumentation: The cytoplasmic asse150
- VI. Nuclear Inclusions Are Formed by Small DNA Viruses152
- A. Adenovirus152
- B. Nuclear inclusions formed during polyomavirus and papillomavirus infection154
- VII. Virus Factories and Inclusions Formed by RNA Viruses155
- A. Reoviruses155
- B. Inclusions formed during arenavirus infection159
- C. Inclusions formed during rabies virus infection159
- VIII. Conclusions160
- References161
- Chapter 5: Parvoviral Host Range and Cell Entry Mechanisms183
- I. Introduction to the Viruses184
- A. The family Parvoviridae184
- B. The genus Parvovirus186
- II. Structure of a Uniquely Dense and Compact Virion188
- A. Rugged 260 Aring protein capsids with T = 1 icosahedral symmetry188
- B. Linear single-stranded DNA genomes with palindromic telomeres190
- C. Creating and expressing transcription templates192
- III. Recognizing the Target: Cell Surface Receptors and Viral Host Range193
- A. The MVM model: Glycan-specific interactions around the twofold symmetry axes195
- B. The FPV/CPV model: Engaging the transferrin receptor at the threefold symmetry axes201
- IV. Breaching the Outer Barrier: To the Cytosol and Beyond205
- A. Structural transitions in the virion induced in vitro206
- B. Essential elements in the VP1-specific entry peptide212
- C. Endocytosis, vacuolar trafficking, and structural transitions in vivo216
- D. From cytosol to nucleus221
- E. Waiting for S-phase: Cryptic versus productive infection223
- Acknowledgments
- References
- Chapter 6: Viral Stress-Inducible Genes233
- List of Abbreviations234
- I. Introduction235
- II. Signaling Pathways Leading to VSIG Induction236
- A. Signaling by dsRNA239
- III. Inhibition of Translation by Proteins Encoded by VSIGs241
- A. The P56 family of proteins242
- B. 2'-5' Oligoadenylate synthetases246
- C. PKR/PACT248
- IV. Viral Evasion of VSIG Expression and Function251
- A. Inhibition of IFN synthesis and VSIG induction253
- B. Inhibition of IFN signaling254
- Acknowledgments256
- References256
- Index265
Published since 1953, Advances in Virus Research covers a diverse range of in-depth reviews providing a valuable overview of the current field of virology.
In 2004, the Institute for Scientific Information released figures showing that the series has an Impact Factor of 2.576, with a half-life of 7.1 years, placing it 11th in the highly competitive category of Virology.
In 2004, the Institute for Scientific Information released figures showing that the series has an Impact Factor of 2.576, with a half-life of 7.1 years, placing it 11th in the highly competitive category of Virology.